Ruptured AAA
Appearance
Definitions:
[edit | edit source]- AAA with extra-luminal blood, either on CT or noted at surgery
- Contained rupture - blood outside the aneurysm sac, but confined to the retroperitoneal space
- Free rupture - bleeding directly into peritoneal cavity
- Symptomatic non-ruptured AAA: back pain or tenderness over the aorta on deep palpation, but with an intact aneurysm on CT/surgery
- The pain is thought to be secondary to acute expansion of the wall, intramural haemorrhage, wall degeneration or bleeding into the thrombus and is considered a prelude to rupture
- Much better prognosis than ruptured, but still require urgent management to prevent rupture
Pathophysiology
[edit | edit source]- RAAA represents failure of the aortic wall to bear the load
- Aortic thrombus plays a role - this impedes blood supply to the wall. Thicker mural thrombus means more likely to rupture.
Pre-op prognosis
[edit | edit source]- Mortality 80-90% overall (30% if they make it to OT)
- Predictors of mortality:
- Hardman index/Glagow aneurysm score, RAAA-POSSUM, Edinburgh Ruptured Aneurysm Score
- None of those is accurate enough to deny repair
- Loss of consciousness - OR 2.6
- Pre-op cardiac arrest - OR 4.3
- Pre-existing CCF
- Pre-existing renal insufficiency
- Advanced age (>76yo) - OR 5.3
- Female
- Suprarenal clamp - OR 2.4
Presentation
[edit | edit source]- Classic triad:
- Hypotension
- Abdo/back pain - typically severe and unrelenting
- Pulsatile abdominal mass
- Frequently appears pale and diaphoretic
- Flank ecchymosis is a late sign
Differential diagnosis
[edit | edit source]- Renal colic
- Diverticulitis
- MI
- Pancreatitis
- GIT bleed
- Perforated ulcer
Investigation:
[edit | edit source]- AXR
- Enlargement of calcified aortic wall beyond normal limits
- Loss of psoas shadow from retroperitoneal haemorrhage
- USS
- FAST
- Excellent for identifying AAA in trained hands
- Not sufficiently accurate to exclude rupture
- FAST
- CT
- Most accurate method for diagnosis, ideally with contrast, to plan open or EVAR
- Do it if the patient is stable
Management
[edit | edit source]Initial management
[edit | edit source]- Immediate vascular consult
- Permissive hypotension - SBP around 80
- Maintain consciousness, minimise organ ischaemia, prevent ST depression
- Be very judicious with fluids. Increased fluids increases mortality. Overall risk of harm is low if the diagnosis is wrong.
- Blood or fluids can be given
- Dual large-bore access
- Art line?
- IDC?
- Crossmatch
- Avoid intubation if they can protect own airway
- CT-A would be very useful if possible - look for periaortic stranding to suggest imminent rupture - however the patient has to be well enough to go to CT
- NGT
Open vs EVAR
[edit | edit source]- EVAR is preferred if possible
- Need a CTA for EVAR - patient has to be stable
- Anatomy is most important (see open AAA repair page for considerations)
- Overall about half of RAAA patients are suitable for EVAR
Operative setup:
[edit | edit source]- Prep, drape THEN intubate
- Consider art line/IDC
- Cell saver
- Avoid hypothermia
Open repair:
[edit | edit source]- Proximal aortic control - laparotomy, retract left lobe of liver and stomach to right, divide gastrohepatic ligament, retract oesophagus to left, access aorta and clamp it.
- Dissect infra-renal aortic neck. The clamp can be repositioned here if there is space
- Tube graft is most commonly used
- Close abdomen primarily if possible. Can't close in 25% due to tension. Early mesh closure may be an option.
EVAR:
[edit | edit source]- Can be done under local + sedation (maintain sympathetic tone) or GA
- Can be either percutaneous access or cutdown
- Stable patients can have GA and a cutdown
- Can do balloon occlusion first
Post-op prognosis:
[edit | edit source]Complications
[edit | edit source]- Often start to go downhill on day 2
- Lower extremity ischaemia
- Either distal embolization or occlusion
- Often kinking or compression of graft if done via EVAR
- Abdominal compartment syndrome
- More common after EVAR, but can also occur after open - esp if significant haemorrhage or oedematous bowel
- Often manifests as renal failure secondary to renal vein compression, or rising peak airway pressures, reduced cardiac output, oliguria
- Decompressive laparotomy for intra-abdominal HTN > 25mmHg
- Ischaemic colitis
- See separate topic under 'open AAA repair'
- Infection
- Graft infection is rare but devastating
- Often requires complete excision of graft
- Groin access complication