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Ruptured AAA

From Surgopaedia

Definitions:

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  • AAA with extra-luminal blood, either on CT or noted at surgery
  • Contained rupture - blood outside the aneurysm sac, but confined to the retroperitoneal space
  • Free rupture - bleeding directly into peritoneal cavity
  • Symptomatic non-ruptured AAA: back pain or tenderness over the aorta on deep palpation, but with an intact aneurysm on CT/surgery
    • The pain is thought to be secondary to acute expansion of the wall, intramural haemorrhage, wall degeneration or bleeding into the thrombus and is considered a prelude to rupture
    • Much better prognosis than ruptured, but still require urgent management to prevent rupture

Pathophysiology

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  • RAAA represents failure of the aortic wall to bear the load
  • Aortic thrombus plays a role - this impedes blood supply to the wall. Thicker mural thrombus means more likely to rupture.

Pre-op prognosis

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  • Mortality 80-90% overall (30% if they make it to OT)
  • Predictors of mortality:
    • Hardman index/Glagow aneurysm score, RAAA-POSSUM, Edinburgh Ruptured Aneurysm Score
    • None of those is accurate enough to deny repair
    • Loss of consciousness - OR 2.6
    • Pre-op cardiac arrest - OR 4.3
    • Pre-existing CCF
    • Pre-existing renal insufficiency
    • Advanced age (>76yo) - OR 5.3
    • Female
    • Suprarenal clamp - OR 2.4

Presentation

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  • Classic triad:
    • Hypotension
    • Abdo/back pain - typically severe and unrelenting
    • Pulsatile abdominal mass
  • Frequently appears pale and diaphoretic
  • Flank ecchymosis is a late sign

Differential diagnosis

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  • Renal colic
  • Diverticulitis
  • MI
  • Pancreatitis
  • GIT bleed
  • Perforated ulcer

Investigation:

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  • AXR
    • Enlargement of calcified aortic wall beyond normal limits
    • Loss of psoas shadow from retroperitoneal haemorrhage
  • USS
    • FAST
      • Excellent for identifying AAA in trained hands
      • Not sufficiently accurate to exclude rupture
  • CT
    • Most accurate method for diagnosis, ideally with contrast, to plan open or EVAR
    • Do it if the patient is stable

Management

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Initial management

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    • Immediate vascular consult
    • Permissive hypotension - SBP around 80
      • Maintain consciousness, minimise organ ischaemia, prevent ST depression
      • Be very judicious with fluids. Increased fluids increases mortality. Overall risk of harm is low if the diagnosis is wrong.
      • Blood or fluids can be given
    • Dual large-bore access
    • Art line?
    • IDC?
    • Crossmatch
    • Avoid intubation if they can protect own airway
    • CT-A would be very useful if possible - look for periaortic stranding to suggest imminent rupture - however the patient has to be well enough to go to CT
    • NGT

Open vs EVAR

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  • EVAR is preferred if possible
  • Need a CTA for EVAR - patient has to be stable
  • Anatomy is most important (see open AAA repair page for considerations)
  • Overall about half of RAAA patients are suitable for EVAR

Operative setup:

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  • Prep, drape THEN intubate
  • Consider art line/IDC
  • Cell saver
  • Avoid hypothermia

Open repair:

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  • Proximal aortic control - laparotomy, retract left lobe of liver and stomach to right, divide gastrohepatic ligament, retract oesophagus to left, access aorta and clamp it.
  • Dissect infra-renal aortic neck. The clamp can be repositioned here if there is space
  • Tube graft is most commonly used
  • Close abdomen primarily if possible. Can't close in 25% due to tension. Early mesh closure may be an option.

EVAR:

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  • Can be done under local + sedation (maintain sympathetic tone) or GA
  • Can be either percutaneous access or cutdown
  • Stable patients can have GA and a cutdown
  • Can do balloon occlusion first

Post-op prognosis:

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Complications

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  • Often start to go downhill on day 2
  • Lower extremity ischaemia
    • Either distal embolization or occlusion
    • Often kinking or compression of graft if done via EVAR
  • Abdominal compartment syndrome
    • More common after EVAR, but can also occur after open - esp if significant haemorrhage or oedematous bowel
    • Often manifests as renal failure secondary to renal vein compression, or rising peak airway pressures, reduced cardiac output, oliguria
    • Decompressive laparotomy for intra-abdominal HTN > 25mmHg
  • Ischaemic colitis
    • See separate topic under 'open AAA repair'
  • Infection
    • Graft infection is rare but devastating
    • Often requires complete excision of graft
  • Groin access complication