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Varicose veins
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== Treatment options == * No treatment * Conservative (compression) ** Generally done as a trial first step for patients in CEAP2-4 (20-30mm Hg) or CEAP6 (30-40mm Hg) ** ESCHAR trial - compression is comparable to compression plus ligation + stripping of the GSV in CEAP6 disease, but ulcer recurrence is much lower with surgery * Surgery ** Rationale and treatment approach: *** Goal is to close refluxing superficial veins *** Treat most proximal point of reflux first - ''most'' (50-70% of patients) have SFJ incompetence, and one study found 85% with VV have GSV reflux and 20% SSV *** Remove diseased segments but leave normal functioning veins behind - not necessary to treat normal or atretic sections of GSV *** Use the least invasive technique possible - start with RFA and step up if necessary *** Need to remove as many superficial varicosities as possible - if left behind, even after GSV ligation +/- stripping, they can drain by alternate pathways and persist. *** GSV disease: RCTs have shown benefit to surgery over conservative management. Early post-operative advantage in pain for endovenous ablation, but no long-term difference. **** Indications for GSV surgery as opposed to EVA: ***** Superficial saphenous tributary (risks skin burns unless 1cm between vein and skin) ***** GSV dilation or aneurysmal venous segments >2.5cm (the probe won't ablate properly because it won't contact the vein walls) ***** Chronic thrombophlebitis (prevents advancement of catheter) ***** Excessive tortuosity (can't advance) ***** Acute superficial thrombophlebitis (EVA contraindicated in any vein with acute thrombus) *** SSV disease: EVA is probably effective, but concern persists over potential for thermal injury to popliteal neurovascular structures. *** Reticular veins/telangiectasias: sclerotherapy - controversial whether this treatment should happen simultaneously with primary VV treatment ** Endovenous *** *** RFA: **** Segmental ablation - catheter contacts vein wall and delivers radiofrequency energy, resulting in destruction of endothelium, contraction of vein wall collagen, and thrombus formation. Eventually fibrosis occurs, resulting in a durable ablation. **** Contraindications: ***** SVT ***** DVT ***** Venous aneurysm ***** ABI < 0.9 ***** Pacemaker - needs assessment by cardiologist prior **** Results ***** 74% had improved CEAP at 36 months ***** 93% had continued occlusion of treated truncal veins at 3 years *** Sclerotherapy **** Introduction of a chemical into a vein to induce endothelial damage that results in thrombosis and eventually fibrosis. **** Indications ***** Most often used to treat smaller vessels such as reticular veins and telangiectasias **** Relative contraindications ***** Asthma ***** Late complications of diabetes ***** Hypercoagulable state ***** Leg oedema ***** Advanced PAD ***** Bad CKD **** Absolute contraindications ***** Known allergy ***** Acute cellulitis ***** Acute respiratory or skin disease ***** Severe systemic disease ***** Phlebitis migrans ***** Acute superficial thrombophlebitis ***** Pregnancy ***** Hyperthyroidism ***** Bedridden status ** Surgical [[Category:Vascular]]
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