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Small bowel obstruction
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== '''Aetiology of jejunal and ileal SBO''' == {| class="wikitable" |'''Extra-luminal''' |Adhesions (65%) | ** Mostly from surgery, but can also be caused by peritonitis, diverticulitis, radiation enteritis, or PID ** Can be congenital ** Likely occurs more for pelvic procedures due to higher mobility of bowel ** Caused by fibrotic bands which are the end result of inflammation at sites of previous injury/surgery |- | |Internal hernia | |- | |Midgut (small bowel) volvulus |A huge load of high-fibre, indigestible food which arrives suddenly in an empty bowel, creating rotational-kinking forces. Uncommon in the developed world. Bowel found to be twisted around an unusually long mesentery, containing litres of clay-like undigested food. Can occur in combination with sigmoid volvulus to form the ileosigmoid knot. Urgent operation. |- | |External hernia (10%) |Typically inguinal or ventral hernia |- | |Neoplastic | ** Carcinomatosis ** Extra-intestinal neoplasm ** Malignancy accounts for about 20% overall - mostly peritoneal carcinomatosis |- | |Intra-abdominal abscess |Commonly ruptured appendix, diverticulitis, or dehiscence of an anastomosis |- |'''Intra-mural''' |Primary tumours (carcinoid, GIST, adenocarcinoma) |Rare |- | |Metastatic tumours (colon, ovarian, gastric, pancreatic, melanoma) | |- | |Malrotation | |- | |Duplications, cysts | |- | |IBD (5%) * Acute inflammation and oedema * Chronic strictures * Also higher risk for malignancy | ** Close discussion with gastroenterologist regarding when surgery is indicated ** Remember Crohn's is incurable, and long-term goal is to minimise length of bowel resected ** Inflammatory strictures may be treatable with steroids ** Fibrotic strictures - adalimumab, endoscopic dilatation ** In these patients, consider nutritional status, immunosuppressants may inhibit wound healing and try to optimise ** 60% of medical treatment of strictures fails and 40% of endoscopic dilatation fails |- | |Infections | ** TB ** Actinomycosis ** Diverticulitis ** Mesenteric adenitis |- | |Traumatic | ** Haematoma ** Ischaemic stricture |- | |Intussusception * Rare in adults (1-5% of SBO) * Benign lesions ** Meckel's ** Polyps * Malignant lesions ** Primary or metastatic tumour * Other ** Anastomotic (e.g. retrograde intussusception of RYGB anastomosis) ** Adhesions ** IBD ** Idiopathic - no lead point 8-20% | ** Usually secondary to a pathological lead-point such as a polyp or tumour ** The lead point is pulled forward by normal peristalsis, resulting in telescoping of the intussusceptum (bowel with lead point) within the intussuscipiens (surrounding normal bowel) ** Malignancy rate varies depending on source - Sabiston says mostly benign in adults, as malignant lesions tend to obstruct via tumour infiltration and adhesions, rather than intussusception. But in the literature, around 60-70% seem to be malignant based on reviews as of 2024. ** Increased incidence along with AIDS - Kaposi, NHL, lymphoid hyperplasia ** Generally, resect. Can start laparoscopic and then in most hands exteriorise the segment through a mini-laparotomy. Perform an oncological resection if in doubt. If benign aetiology suspected, can reduce the intussusception prior to resection, to decrease the length of resection. |- | |Endometriosis | |- | |Radiation enteropathy/stricture | |- |'''Intra-luminal''' |Gallstone | |- | |Enterolith |Often originating from a jejunal diverticulum - see separate topic |- | |Foreign body/bezoar * Phytobezoars (vegetables or fruits - persimmons, popcorn) * Trichobezoars (hair) * Parasitic (ascaris lumbricoides in distal ileum) | |}
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